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The diagnosis of hcm is confirmed with the presence of a left ventricular wall thickness of ≥15 mm that is otherwise unexplained by abnormal loading conditions (e.g., hypertension, valvular, congenital disease) or infiltrative cardiomyopathies. Diagnosis of hcm relies on echocardiogram and cardiac mri showing hypertrophied left ventricle (lv) without dilatation in the absence of other metabolic or systemic disease causing hypertrophy such as hypertension or valvular disease. Diagnosis typically involves an echocardiogram to visualize the thickened septum and assess heart function

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Other tests may include an ecg, mri, or genetic testing. 1,2 this module describes the information required from echocardiography to diagnose heart valve disease and inform decisions about further management. The criteria for diagnosing asymmetric septal hypertrophy is septal thickness greater than or equal to 15 mm (normal wall thickness is 12 mm or less, measured during diastole) or ratio of septal thickness to the thickness of the left ventricular inferior.

Since there is a thickened septum blood flow is high and turbulent through the lvot

The high flow of blood through the lvot can draw the mitral valve anteriorly and towards the lv outflow track causing more obstruction This is known as the ventruri effect. Hypertrophic cardiomyopathy (hcm) is a type of cardiomyopathy defined by left ventricular hypertrophy which cannot otherwise be explained by another cardiac or systemic disease It is the leading cause of sudden cardiac death in infants, teenagers, and young adults.

The mainstay of diagnosis in heart valve disease is imaging, using transthoracic echocardiography

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